A new study, published in Environmental Health Perspectives, suggests that exposure to ultrafine particles (UFP) of air pollution can be a trigger for heart attacks.
To investigate the link between UFP and nonfatal heart attacks, the team of researchers analyzed data from air pollution monitoring sites of Augsburg in Germany from 2005 to 2015. They also compared these data with the cases of nonfatal heart attacks in the city during the same period.
The first author of the study Kai Chen, Ph.D., assistant professor at Yale School of Public Health in New Haven, CT, says: “This study confirms something that has long been suspected — air pollution’s tiny particles can play a role in serious heart disease. This is particularly true within the first few hours of exposure. Elevated levels of UFP are a serious public health concern.”
A new study, conducted by the researchers from Aarhus University in Denmark, suggests that exposure to air pollution in childhood may increase the risk of developing schizophrenia.
For the study, the scientists analyzed data received from 23,355 people who were born from May 1, 1981, till December 31, 2002. Their evolution was followed up from the participants 10th birthday to the first occurrence of schizophrenia, emigration, death or December 31, 2012, whichever happened first.
The analysis showed that people who were exposed to high levels of air pollution during their childhood had a higher risk of schizophrenia later in life.
Senior researcher Henriette Thisted Horsdal, Ph.D., says: The study shows that the higher the level of air pollution, the higher the risk of schizophrenia. For every 10 micrograms per cubic meter [referring to the concentration of the pollutant nitrogen dioxide in ambient air] increase in the daily average, the risk of schizophrenia increases by approximately 20%.”
New research from the Future Science Research Centre in the Republic of Korea suggests that air pollution may have a great impact on hair loss.
For their research, a team of scientists exposed human follicle dermal papilla cells (HFDPC) to particles of dust and diesel of size PM10, which diameter is generally 10 micrometer and smaller. The analysis of the gathered data showed that exposure to PM10-like particles lowered levels of a protein key for hair growth.
Lead author of the study Hyuk Chul Kwon says: “Our research explains the mode of action of air pollutants on [HFDPCs], showing how the most common air pollutants lead to hair loss.”
A new study, published in the Nature Communications, finds that black carbon particles can get into the part of the placenta that feeds the developing fetus.
To check the influence of the soot, a team of researchers examined placentas from 5 pre-term and 23 full-term births. With the help of high-resolution imaging, the scientists detected particles of black carbon in the fetal side of every placenta taken for examination.
Of all the participants, 10 mothers who lived in high traffic areas and were exposed to the highest levels of pollution during pregnancy had the highest levels of particles in the placenta.
The scientists write in their paper: “Our results demonstrate that the human placental barrier is not impenetrable for particles. Further research will have to show whether the particles cross the placenta and reach the fetus.”
A new study of Chinese adults finds a link between air pollution and a higher risk of developing atherosclerosis, or coronary artery calcification, as well as death from heart disease.
To investigate the influence of air pollution, a team of researchers analyzed data on 8,867 Chinese people whose age varied from 25 to 92 years. All of them had suspected coronary heart disease and were recruited between 2015 and 2017.
The analysis showed that for each nitrogen dioxide increase of 20 μg/m3, the risk of having a high coronary artery calcium score increased by 24.5 per cent, and for each 30 μg/m3, the risk increased by 27.2 per cent.
The lead author Meng Wang says: “This study may provide evidence that coronary atherosclerosis is a pathological pathway through which air pollution exposure increases risk of death from coronary heart disease.”
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