A recent study from the US finds a strong association between ambient air pollution and increased rates of emphysema, chronic respiratory disease.
A team of researchers, led by Joel D. Kaufman, MD, MPH, of the School of Public Health at University of Washington, and R. Graham Barr, MD, DrPH, of the Department of Medicine at Columbia University Medical Center, used data from the Multi-Ethnic Study of Atherosclerosis (MESA) which included patients aged 45 to 84 years from 6 metropolitan areas in the US.
Researchers found that median percent emphysema was 3% at the beginning of the study, then increased a mean 0.58 percentage points per 10 years. Mean ambient concentrations of PM2.5 and NOx decreased substantially during the follow-up period. Concentrations of each of the observed ambient pollutant estimates were associated with increases in percent emphysema per 10 years.
A new research, published in the European Respiratory Journal, finds a diet rich in tomatoes may restore lung function in people who quit smoking and slow down lung function decline and all people.
In the course of the study, the scientists found that those adults who ate more than three portions of fruit or more than two tomatoes experience slower lung function decline than those who consumed fewer than one serving of fruit or one tomato a day.
The lead study author Vanessa Garcia-Larsen, an assistant professor in the Bloomberg School’s Department of International Health, says: “This study shows that diet might help repair lung damage in people who have stopped smoking. It also suggests that a diet rich in fruits can slow down the lung’s natural aging process even if you have never smoked.”
The scientists hope that this drug, inspired by an antibody found in the sharks’ blood can treat the condition successfully. They can start human trials already within the next year.
Dr Mick Foley, one of the researchers from the La Trobe Institute for Molecular Science, says: “Fibrosis is the end result of a lot of different insults and injuries. This molecule can kill the cells that cause fibrosis.”
A team of researchers from the University of Southern California finds that teenage users of e-cigarettes are twice more likely to get bronchitis compared to children who have never smoked electronic cigarettes.
For their study, the scientists analyzed the responses from more than 2,000 older teenagers, asking for symptoms of chronic bronchitis such as a daily cough for three months straight.
Researchers found that those teenagers who vape had 71% higher risk of the condition which was connected to the risk of damaging lungs.
Dr Rob McConnell, a professor of preventive medicine at the Keck School of Medicine, within the University of Southern California, says: “E-cigarettes are known to deliver chemicals toxic to the lungs, including oxidant metals, glycerol vapour, diketone flavouring compounds and nicotine. However, there has been little study of the chronic health effects of e-cigarettes.”
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